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Arthritis Pain

About Osteoarthritis (OA) Pain

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Osteoarthritis (OA) prevalence is high – about 10% of people over 60 years of age have symptomatic OA, rising to 30%+ by age 80, with the knees, then hips, hands, and spine most commonly affected. OA pain arises from ligament instability and cartilage breakdown, triggering multiple mechanisms: periosteal irritation from osteophytes, synovitis activating nociceptors, increased subchondral bone pressure, joint effusion stretching the capsule, muscle fatigue/spasm, and central sensitization amplifying signals. OA differs from Rheumatoid Arthritis (RA), whereas RA is an autoimmune attack on the joint lining with usually symmetrical joint involvement, initially starting in the hands and feet and then progressing to larger joints such as the shoulders, elbows, knees, ankles, hips and cervical spine. RA symptoms usually include prolonged morning stiffness (>1hr), systemic fatigue and a low-grade fever from the chronic inflammation.

Symptoms

Deep, achy use-related pain (worse with activity, better at rest), morning/post-inactivity stiffness (<30 min), tenderness, grating/crepitus, swelling, reduced range of motion, bone spurs, joint instability, and occasional night pain in advanced cases.

Known Causes

  • Primary: Ligament instability causing mechanical joint stress/overuse and age-related cartilage wear, obesity increasing load.
  • Secondary: Prior spinal or joint injury/trauma, congenital deformities (e.g., hip dysplasia), repetitive microtrauma (athletes), inflammatory diseases (e.g., post-septic arthritis), metabolic disorders (e.g., hemochromatosis), and genetic predisposition (e.g., collagen defects), along with nutrient deficiencies, systemic toxicities or infections.
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